Hard Tissue Growth, Repair and Remineralization; Symposium by Ciba Foundation;

By Ciba Foundation;

Chapter 1 Chairman's creation (pages 3–5): R. F. Sognnaes
Chapter 2 Mineral Regeneration in Echinoderms and Molluscs (pages 7–33): Karl M. Wilbur
Chapter three Remineralization of teeth (pages 35–56): D. F. G. Poole and L. M. Silverstone
Chapter four Chemical brokers within the regulate of Calsification techniques in organic structures (pages 57–90): Marion D. Francis, William W. Briner and John A. Gray
Chapter five the present prestige of Allogeneic enamel Transplantation: Attachment fix, Regeneration and improvement in Dental Replants, Transplants and Implants (pages 91–119): Leonard B. Shulman
Chapter 6 Induction of Bone fix through a number of Bone?Grafting fabrics (pages 121–141): Philip J. Boyne
Chapter 7 Enzymes in Bone Morphogenesis: Endogenous Enzymic Degradation of the Morphogenetic estate in Bone in strategies Buffered by means of Ethylenediaminetetraacetic Acid (EDTA) (pages 143–160): Marshall R. Urist
Chapter eight Chairman's creation (pages 163–167): Dame Janet M. Vaughan
Chapter nine made up our minds and Inducible Osteogenic Precursor Cells (pages 169–185): A. J. Friedenstein
Chapter 10 An Ultrastructural research of Calcium and Phosphate Deposition and trade in Tissues (pages 187–211): J. L. Matthews, J.h. Martin, J. W. Kennedy Iii and E. J. Collins
Chapter eleven Calcium?Accumulating Vesicles within the Intercellular Matrix of Bone (pages 213–246): H. Clarke Anderson
Chapter 12 Biochemistry of Collagens from Mineralized Tissues (pages 247–261): M. J. Barnes
Chapter thirteen Albumin in Bone (pages 263–293): Maureen Owen, J. T. Triffitt and R. A. Melick
Chapter 14 Lysosomal Enzymes in Skeletal Tissues (pages 295–313): J. T. Dingle
Chapter 15 Bone Remodelling: in vitro experiences on nutrition D Metabolites (pages 315–330): John J. Reynolds
Chapter sixteen the results of Pyrophosphate and Diphosphonates on Calcium Metabolism (pages 331–358): H. Fleisch, R. G. G. Russell, S. Bisaz and J.P. Bonjour
Chapter 17 nutrition D and the Skeleton (pages 359–371): E. Kodicek
Chapter 18 Calcium Absorption and a few houses of the nutrition D?Dependent Calcium?Binding Protein (pages 373–389): R.h. Wasserman
Chapter 19 Renal and Skeletal interplay: The function of diet D (pages 391–408): S. W. Stanbury, L. F. Hilland and E. B. Mawer
Chapter 20 Plasma Calcium Homeostasis (pages 409–438): M. Peacock and B. E. C. Nordin
Chapter 21 Chairman's Concluding comments (pages 439–441): R. F. Sognnaes
Chapter 22 Addendum on Fluorides (pages 442–443): R. F. Sognnaes

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9) ; R, surface ‘conditioned’ by exposure to a mineralizing solution for 4 days. 0); yP = pg phosphorus dissolved; yE = pg enamel dissolved. Total depth dissolved after 50 etches, 71 pm (C), 39 pm (R). Thus ‘conditioning’ with a mineralizing solution results in a significant reduction in the solubility of enamel. REMINERALIZATION OF ENAMEL 45 by exposure to remineralizing solutions resulted in an increased resistance to artificial caries (Silverstone 1971) associated with a decreased surface solubility (Figs.

Moreover, not only is it difficult to distinguish hydroxyapatite from octacalcium phosphate by means of electron diffraction, but both octacalcium phosphate and dicalcium phosphate are fairly easily hydrolysed to hydroxyapatite. Even X-ray diffraction studies fail to distinguish between intact demineralized and remineralized enamel (Feagin et al. 1970). 1) as normal enamel (Wei 1970). Recent measurements on various parameters of the model remineralizing system show that fluoride not only increases the rate of deposition of calcium phosphate but also becomes incorporated into the mineral, probably as fluorapatite or fluorohydroxyapatite (Feagin 1971).

Once established, dental caries is likely to persist and the principal aim must therefore be to prevent the disease from starting in the first place. Prolonged searches have therefore been made for substances which are known not only to contribute to the protection of the tooth surfaces but also to stand a reasonable chance of actually penetrating the plaque and reaching surfaces at risk. In this respect the marked reducing effect on caries of the fluoride ion has attracted most attention, yet, even now, the exact way in which it acts to reduce caries is in doubt.

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